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  • Source: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidade: IQ

    Subjects: ESTATINAS, LIPOPROTEÍNAS HDL, PROTEÔMICA

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    • ABNT

      RONSEIN, Graziella Eliza et al. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 41, p. 2330–2341, 2021Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.121.316278. Acesso em: 01 maio 2024.
    • APA

      Ronsein, G. E., Vaisar, T., Davidson, W. S., Bornfeldt, K. E., Probstfield, J. L., O’Brien, K. D., et al. (2021). Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects. Arteriosclerosis, Thrombosis, and Vascular Biology, 41, 2330–2341. doi:10.1161/ATVBAHA.121.316278
    • NLM

      Ronsein GE, Vaisar T, Davidson WS, Bornfeldt KE, Probstfield JL, O’Brien KD, Zhao X-Q, Heinecke JW. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2021 ; 41 2330–2341.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/ATVBAHA.121.316278
    • Vancouver

      Ronsein GE, Vaisar T, Davidson WS, Bornfeldt KE, Probstfield JL, O’Brien KD, Zhao X-Q, Heinecke JW. Niacin increases atherogenic proteins in high-density lipoprotein of statin-treated subjects [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2021 ; 41 2330–2341.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/ATVBAHA.121.316278
  • Source: Molecular Metabolism. Unidades: IME, FCF

    Assunto: ARTERIOSCLEROSE

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    • ABNT

      SCOLARO, Bianca et al. Statin dose reduction with complementary diet therapy: apilot study of personalized medicine. Molecular Metabolism, v. 11, p. 137-144, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2018.02.005. Acesso em: 01 maio 2024.
    • APA

      Scolaro, B., Nogueira, M. S., Paiva, A. C. de, Bertolami, A., Barroso, L. P., Vaisar, T., et al. (2018). Statin dose reduction with complementary diet therapy: apilot study of personalized medicine. Molecular Metabolism, 11, 137-144. doi:10.1016/j.molmet.2018.02.005
    • NLM

      Scolaro B, Nogueira MS, Paiva AC de, Bertolami A, Barroso LP, Vaisar T, Heffron SP, Fisher EA, Castro IA de. Statin dose reduction with complementary diet therapy: apilot study of personalized medicine [Internet]. Molecular Metabolism. 2018 ; 11 137-144.[citado 2024 maio 01 ] Available from: https://doi.org/10.1016/j.molmet.2018.02.005
    • Vancouver

      Scolaro B, Nogueira MS, Paiva AC de, Bertolami A, Barroso LP, Vaisar T, Heffron SP, Fisher EA, Castro IA de. Statin dose reduction with complementary diet therapy: apilot study of personalized medicine [Internet]. Molecular Metabolism. 2018 ; 11 137-144.[citado 2024 maio 01 ] Available from: https://doi.org/10.1016/j.molmet.2018.02.005
  • Source: Current Opinion in Lipidology. Unidade: IQ

    Subjects: COLESTEROL, INFLAMAÇÃO, DOENÇAS CARDIOVASCULARES

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      RONSEIN, Graziella Eliza e VAISAR, Tomas. Inflammation, remodeling, and other factors affecting HDL cholesterol efflux. Current Opinion in Lipidology, v. 28, n. 1, p. 52-59, 2017Tradução . . Disponível em: https://doi.org/10.1097/MOL.0000000000000382. Acesso em: 01 maio 2024.
    • APA

      Ronsein, G. E., & Vaisar, T. (2017). Inflammation, remodeling, and other factors affecting HDL cholesterol efflux. Current Opinion in Lipidology, 28( 1), 52-59. doi:10.1097/MOL.0000000000000382
    • NLM

      Ronsein GE, Vaisar T. Inflammation, remodeling, and other factors affecting HDL cholesterol efflux [Internet]. Current Opinion in Lipidology. 2017 ; 28( 1): 52-59.[citado 2024 maio 01 ] Available from: https://doi.org/10.1097/MOL.0000000000000382
    • Vancouver

      Ronsein GE, Vaisar T. Inflammation, remodeling, and other factors affecting HDL cholesterol efflux [Internet]. Current Opinion in Lipidology. 2017 ; 28( 1): 52-59.[citado 2024 maio 01 ] Available from: https://doi.org/10.1097/MOL.0000000000000382
  • Source: Jcl insight. Unidade: IQ

    Subjects: COLESTEROL, MACRÓFAGOS, APOLIPOPROTEÍNAS

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      PAMIR, Nathalie et al. Plasminogen promotes cholesterol efflux by the ABCA1 pathway. Jcl insight, v. 2, n. 15, p. 1-15 art. 92176, 2017Tradução . . Disponível em: https://doi.org/10.1172/jci.insight.92176. Acesso em: 01 maio 2024.
    • APA

      Pamir, N., Hutchins, P. M., Ronsein, G. E., Wei, H., Tang, C., Das, R., et al. (2017). Plasminogen promotes cholesterol efflux by the ABCA1 pathway. Jcl insight, 2( 15), 1-15 art. 92176. doi:10.1172/jci.insight.92176
    • NLM

      Pamir N, Hutchins PM, Ronsein GE, Wei H, Tang C, Das R, Vaisar T, Plow E, Schuster V, Reardon CA, Weinberg R, Dichek DA, Marcovina S, Getz GS, Heinecke JW. Plasminogen promotes cholesterol efflux by the ABCA1 pathway [Internet]. Jcl insight. 2017 ; 2( 15): 1-15 art. 92176.[citado 2024 maio 01 ] Available from: https://doi.org/10.1172/jci.insight.92176
    • Vancouver

      Pamir N, Hutchins PM, Ronsein GE, Wei H, Tang C, Das R, Vaisar T, Plow E, Schuster V, Reardon CA, Weinberg R, Dichek DA, Marcovina S, Getz GS, Heinecke JW. Plasminogen promotes cholesterol efflux by the ABCA1 pathway [Internet]. Jcl insight. 2017 ; 2( 15): 1-15 art. 92176.[citado 2024 maio 01 ] Available from: https://doi.org/10.1172/jci.insight.92176
  • Source: Circulation Research. Unidade: IQ

    Subjects: APOLIPOPROTEÍNAS, ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES

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      MONETTE, Jeffrey S et al. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration. Circulation Research, v. 119, n. 1, p. 83-90, 2016Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.116.308357. Acesso em: 01 maio 2024.
    • APA

      Monette, J. S., Hutchins, P. M., Ronsein, G. E., Wimberger, J., Irwin, A. D., Tang, C., et al. (2016). Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration. Circulation Research, 119( 1), 83-90. doi:10.1161/CIRCRESAHA.116.308357
    • NLM

      Monette JS, Hutchins PM, Ronsein GE, Wimberger J, Irwin AD, Tang C, Sara JD, Shao B, Vaisar T, Lerman A, Heinecke JW. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration [Internet]. Circulation Research. 2016 ; 119( 1): 83-90.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/CIRCRESAHA.116.308357
    • Vancouver

      Monette JS, Hutchins PM, Ronsein GE, Wimberger J, Irwin AD, Tang C, Sara JD, Shao B, Vaisar T, Lerman A, Heinecke JW. Patients with coronary endothelial dysfunction have impaired cholesterol efflux capacity and reduced HDL particle concentration [Internet]. Circulation Research. 2016 ; 119( 1): 83-90.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/CIRCRESAHA.116.308357
  • Source: Arteriosclerosis Thrombosis and Vascular Biolology. Unidade: IQ

    Subjects: ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES, MACRÓFAGOS

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    • ABNT

      RONSEIN, Graziella Eliza et al. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects. Arteriosclerosis Thrombosis and Vascular Biolology, v. 36, n. 2, p. 404-411, 2016Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.115.306268. Acesso em: 01 maio 2024.
    • APA

      Ronsein, G. E., Hutchins, P. M., Isquith, D., Vaisar, T., Zhao, X. -Q., & Heinecke, J. W. (2016). Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects. Arteriosclerosis Thrombosis and Vascular Biolology, 36( 2), 404-411. doi:10.1161/ATVBAHA.115.306268
    • NLM

      Ronsein GE, Hutchins PM, Isquith D, Vaisar T, Zhao X-Q, Heinecke JW. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects [Internet]. Arteriosclerosis Thrombosis and Vascular Biolology. 2016 ; 36( 2): 404-411.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/ATVBAHA.115.306268
    • Vancouver

      Ronsein GE, Hutchins PM, Isquith D, Vaisar T, Zhao X-Q, Heinecke JW. Niacin therapy increases high-density lipoprotein particles and total cholesterol efflux capacity but not ABCA1-specific cholesterol efflux in statin-treated subjects [Internet]. Arteriosclerosis Thrombosis and Vascular Biolology. 2016 ; 36( 2): 404-411.[citado 2024 maio 01 ] Available from: https://doi.org/10.1161/ATVBAHA.115.306268
  • Source: Journal of Lipid Research. Unidade: IQ

    Subjects: ARTERIOSCLEROSE, DOENÇAS CARDIOVASCULARES, APOLIPOPROTEÍNAS, LIPOPROTEÍNAS

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      PAMIR, Nathalie et al. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway. Journal of Lipid Research, v. 57, n. 2, p. 246-257, 2016Tradução . . Disponível em: https://doi.org/10.1194/jlr.M063701. Acesso em: 01 maio 2024.
    • APA

      Pamir, N., Hutchins, P., Ronsein, G. E., Vaisar, T., Reardon, C. A., Getz, G. S., et al. (2016). Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway. Journal of Lipid Research, 57( 2), 246-257. doi:10.1194/jlr.M063701
    • NLM

      Pamir N, Hutchins P, Ronsein GE, Vaisar T, Reardon CA, Getz GS, Lusis AJ, Heinecke JW. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway [Internet]. Journal of Lipid Research. 2016 ; 57( 2): 246-257.[citado 2024 maio 01 ] Available from: https://doi.org/10.1194/jlr.M063701
    • Vancouver

      Pamir N, Hutchins P, Ronsein GE, Vaisar T, Reardon CA, Getz GS, Lusis AJ, Heinecke JW. Proteomic analysis of HDL from inbred mouse strains implicates APOE associated with HDL in reduced cholesterol efflux capacity via the ABCA1 pathway [Internet]. Journal of Lipid Research. 2016 ; 57( 2): 246-257.[citado 2024 maio 01 ] Available from: https://doi.org/10.1194/jlr.M063701

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